[Asn23]-beta-Amyloid (1-40), Iowa Mutation – 0.5 mg

Overview

The [Asn23]-beta-Amyloid (1-40), Iowa Mutation is a key peptide for researchers studying neurodegenerative diseases, particularly Alzheimer’s Disease. This specific mutation occurs when the amino acid aspartic acid at position 23 is replaced by asparagine, leading to a more aggressive aggregation of beta-amyloid into toxic fibrils. Understanding the role of this mutated peptide can provide crucial insights into the mechanisms underlying Alzheimer’s and cerebral amyloid beta-protein angiopathy (CAA).

Key Features

• Designed for research on Alzheimer’s Disease and related conditions
• Rapid aggregation capabilities enhance the study of toxic fibril formation
• High purity and quality for reliable results
• Available in a convenient 0.5 mg package
• Supported by extensive data on the Iowa mutation and its implications

Specifications

FAQ

What is the significance of the Iowa Mutation in Alzheimer’s research?

The Iowa Mutation is linked to a higher propensity for beta-amyloid to aggregate into toxic fibrils, which is a hallmark of Alzheimer’s pathology. Studying this peptide helps elucidate the pathogenic mechanisms of Alzheimer’s Disease.

How should I store the [Asn23]-beta-Amyloid peptide?

For optimal stability, store the peptide at -20°C. Avoid repeated freeze-thaw cycles to maintain its integrity.

Can I use this peptide for in vitro studies?

Yes, the [Asn23]-beta-Amyloid (1-40), Iowa Mutation is suitable for various in vitro experiments aimed at understanding amyloid aggregation and neurotoxicity.

If you’re looking to further your research in neurodegenerative diseases, consider exploring other related products such as PKG Substrateor Autocamtide-2. Enhance your studies with our range of high-quality peptides and detection tools to achieve comprehensive results.

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