Bak BH3 (72-87) – 1 mg

Overview

Bak BH3 (72-87) peptide is derived from the BH3 domain of Bak, known as Flu-BakBH3. This peptide exhibits high-affinity binding to a critical surface pocket on the Bcl-XL protein. Bcl-XL plays a vital role as a death antagonist, which is essential in regulating apoptosis. The specific interaction between Bak BH3 and Bcl-XL is crucial for understanding cellular responses to stress and apoptosis induction. If you are studying apoptosis, our Bak BH3 peptide is a key reagent to include in your experiments.

Key Features

• Highly specific for Bcl-XL binding
• Essential for death antagonist function
• Provides reliable results in apoptosis studies
• Available in a convenient 1 mg size
• Compatible with various labeling technologies

Specifications

FAQ

What is the role of Bak BH3 in apoptosis?

Bak BH3 functions as a pro-apoptotic factor by binding to Bcl-XL, which inhibits or prevents cell death. This interaction is a critical part of the regulatory mechanisms controlling apoptosis.

How does the high-affinity binding improve research outcomes?

The high-affinity binding of Bak BH3 to Bcl-XL enhances the precision in studies of apoptosis by ensuring that the peptide effectively interacts with the target protein, which can lead to more reliable data.

Can I use this peptide in conjunction with other assays?

Absolutely! Bak BH3 can be utilized in various experiments, including cell viability assays and fluorescence studies. Consider pairing it with our other peptide options for comprehensive research, such as PKG Substrate or Autocamtide-2.

For those involved in detailed apoptosis studies, adding our Bak BH3 (72-87) peptide to your toolkit will provide you with a reliable method to investigate Bcl-XL interactions. Reach out to our team today to order this essential peptide and enhance your research capabilities.

Related Products

• CREBtide [KRREILSRRPSYR], 5-FAM labeled

Browse more in Peptides.